Studies have shown that excess cholesterol in the blood stream can cause major damage to your cardiovascular system, resulting in various forms of heart disease and even death. All of your cells need cholesterol, yet they have only a limited ability to produce it. In order to get cholesterol where it is needed, the liver has developed a system that puts cholesterol and and triglycerides together into lipoproteins. There are three lipoproteins in your bloodstream, very low density lipoprotein (VLDL), low density lipoprotein (LDL) and high density lipoprotein (HDL) Two of these are produced by your liver (VLDL and HDL) while LDL is created in the bloodstream from VLDL under certain circumstances. VLDL carries triglycerides to the tissues and cells and unloads some which is used for energy or stored as fat.VLDL, however, still has cholesterol, which is used for hormone production, nerve insulation, cell wall creation, to name a few.
If your body produces too much cholesterol, or if you eat too much of the wrong foods, you would have more cholesterol than is needed by the body and it would eventually start depositing on the artery walls. Cholesterol passes through the artery wall quite easily, since the membrane covering the inner surface of every artery is made of fat. The cells of the artery wall shed chemical wastes that can cause oxidation of the trapped LDL cholesterol. Substances known as antioxidants serve as preservatives and keep the LDL from spoiling. If antioxidants are present in the LDL itself, or if it is present in the area where LDL is trapped they can prevent this oxidation from taking place.
Vitamin E, a fat-soluble antioxidant is one of the most effective in preventing this oxidation. There are many other antioxidants as well. Iron, on the other hand, stimulates the oxidation of LDL. Once this oxidation of LDL occurs it attracts other cells into the space it occupies in the artery wall. These cells eat the spoiling rancid LDL and become "foam cells" Foam cells fill up with cholesterol and ultimately burst open when they die. In the process, various factors stimulate the growth of smooth muscle cells in the artery wall, which means that there is an overgrowth of muscle. Over a number of years, all of these cellular events contribute to the progress of atherosclerosis.
It's easy to see why LDL is called the "bad Cholesterol".It is responsible for the formation of plaque in the arterial wall. This can lead to atherosclerosis and other debilitating conditions. What then is "good cholesterol"?HDL carries cholesterol back to the liver. HDL is produced principally by the liver and intestines and carries very little fat. It is the most soluble and easily transported of the lipoproteins.
HDL is known as the good cholesterol for good reason.HDL is able to tow VLDL back to the liver before they can be converted into LDL. HDL can also inhibit the LDL oxidation process, thus preventing the cholesterol from going rancid.
You should reduce your overall fat and calorie consumption to minimize the amount of LDL circulating in your blood stream. However, you should also take positive steps to increase your HDL level independently of your cholesterol count. Losing weight can lead to a decrease in triglycerides and an increase in HDL cholesterol. Giving up smoking has a number of benefits, including a reduction in LDL oxidation. As mentioned before, Vitamin E can help prevent oxidation of LDL as well. Exercising can increase HDL and lower total cholesterol.
In the battle to stop atherosclerosis, multiple approaches work best. There is no single magic bullet. Natural therapies only produce the greatest improvement when used in combination with a healthy diet and exercise. In fact the most powerful cholesterol lowering drugs can be neutralised by poor diets and lifestyles.
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